Energy Shot BoxEnergy Shot BoxEnergy Shot BoxEnergy Shot Box

Phoenix Energy Shots

$34.99

Reviews

  • Three Products In One!
  • Mood • Focus • Energy
  • 12 Active Ingredients
  • Transparent Label - No Fillers
  • Premium Nootropic Blend
  • 12 Shots Per Box

  • The Science
  • Sources
  • FAQs
What is Energy? The simplest definition is "the ability to do work". For a long time that's been the only expectation of what an "energy" supplement is supposed to provide, be it your coffee, energy drink, shot, or pills. At Phoenix Energy, we feel that expectation is extremely outdated. Real energy comes not only from your body, but also your mind and mood and that's exactly what Phoenix Energy's 3-IN-1 Formula was designed to provide.  

Our Patent Pending Formula was designed relying on the lastest in scientific research and study. Through that evolved a formulation of TWELVE active ingredients within three separate product blends designed to Elevate your MOOD, Enhance your FOCUS, and provide a long lasting Boost in ENERGY...ALL IN ONE SHOT!

We proudly display our entire list of proven ingredients, including their doses – NO PROPRIETARY BLENDS and NO FILLERS, so you know exactly what you’re getting.

Take your Energy to a new level and RISE UP with Phoenix Energy!

ENERGY BLEND - 1,381mg

Citrulline Malate 2:1 (1,000mg)

First, Citrulline Malate is considered one the most effective methods for increasing body arginine levels which is then converted to nitric oxide, increasing vasodilation for better nutrient and oxygen delivery to working cells, such as brain cells. Second, supplying the malate fosters more efficient ATP production. The two effects working together create a better metabolic environment for cellular function.

  • Citrulline malate supplementation has been shown to reduce fatigue, improve memory, and increase metabolic efficiency.
  • Supplementing with citrulline has been shown to increase NO synthesis nearly 10-fold (Kim et al., 2015)
  • By restoring nitric oxide function and enhancing cellular energy supply, citrulline malate can improve brain cell health and promote memory (Yabuki et al., 2013)

 

Caffeine Anhydrous (280mg)

Caffeine is the active ingredient in coffee, tea, and other beverages used to reduce tiredness and promote alertness. Caffeine works by stimulating a release of epinephrine (adrenaline) and blocking adenosine. Epinephrine, a neurotransmitter, helps increase vigor and attention, while adenosine antagonization promotes wakefulness. In Phoenix Energy, caffeine is combined with L-Theanine, discussed below.

  • Caffeine anhydrous is simply a dried, powdered version of caffeine. Anhydrous means “without water.”
  • Taking caffeine has been proven to increase feelings of arousal (wakefulness), positive mood, reaction time, and memory (Childs & de Wit, 2006, Gupta, 1988).
  • Supplementing caffeine improves task performance and focus while reducing feelings of mental fatigue (Kennedy and Scholey, 2004)

Dynamine® (100mg)

Dynamine® is poised to disrupt the energy category with its backed-by-science benefits.  A purine alkaloid found in the kucha tea leaf known as Methylliberine. It has been shown to amplify feelings of energy, mood and focus by activating dopamine receptors and other key neurotransmitters, inhibiting adenosine receptors, all without elevating heart rate or blood pressure.

  • Theacrine has been shown to enhance mood, vigor, and cognition. Dynamine shares a similar chemical structure with theacrine.
  • May be capable of increasing dopamine, thereby boosting mood and even feelings of euphoria.
  • Has the potential to work synergistically with caffeine, creating greater enhancements in energy and mood (Kuhman, Joyner, and Bloomer, 2015).
  • Clinically shown to increase the positive benefits of caffeine TWO-FOLD!! (Compound Solutions, Inc., 2020)

 

Rauwolscine (1.5mg)

Rauwolscine is an alkaloid also known as alpha-yohimbine, with which it shares many effects. It is an adrenal system augmenter, which helps with neurotransmitter release. In doing so, rauwolscine helps with sensations of energy as well as enhancing effects of other ingredients in Phoenix Energy. Rauwolscine is found in the rauwolfia genus of plants.

  • One of the functions of rauwolscine is to reduce stimulation of alpha-2 adrenergic receptors. This potentiates effects of other stimulants, such as caffeine.
  • Another effect of rauwolscine supplementation is optimized serotonin – a major neurotransmitter involved in good mood.
  • Supplementation with rauwolscine has been noted to reduce anxiety, including social anxiety.

MOOD BLEND - 755mg

DL-Phenylalanine (400mg)

Phenylalanine is an essential amino acid. It is also a precursor to neurotransmitters, dopamine, and noradrenaline. These two neurotransmitters are critical for mood enhancement and vitality.

  • By increasing dopamine levels, phenylalanine supplementation is useful for reducing feelings of depression and promoting good mood.
  • Adding phenylalanine has been shown to reduce instances of anger, feelings of restlessness, and reports of poor concentration (Wood, Reimherr, and Wender, 1985).
  • In those reporting symptoms of depression, DL-phenylalanine supplementation eliminated these feelings in 67% of participants (Fischer et al., 1975, Beckmann, Strauss, and Ludolph, 1977)

 

N-Acetyl L-Tyrosine (250mg)

Like phenylalanine, tyrosine is an amino acid. In the N-Acetyl form, L-Tyrosine provides a better nootropic effect. Tyrosine helps activate the “fight or flight” response via the creation of key neurotransmitters. Often, when we feel like we cannot concentrate it is a result of “using up” these neurotransmitters, and N-Acetyl L-Tyrosine works to correct such an imbalance.

  • Tyrosine supplementation improves mental acuity, mood, and focus, by increasing catecholamine levels.
  • Tyrosine shines during periods of stress, sleep deprivation, and other times when energy and vigor are compromised.
  • A study in participants who were deprived of one whole night’s sleep found that L-tyrosine supplementation served to counteract the negative effects of the extended wake period (Neri et al., 1995)

 

L-Theanine (90mg)

L-Theanine is a relatively new discovery in the nootropic and mood space. Found in tea, theanine is an amino acid which acts as a GABA mimetic. GABA is a “downer” neurotransmitter that works to mitigate feelings of stress and anxiety. While many love the effects of caffeine, the most common side effect is anxiety. L-Theanine nullifies caffeine’s effects of anxiety, if present, and bolsters its effects on mood and concentration.

  • Despite its “downer” effects, L-Theanine does not cause drowsiness or have sedative-like effects. It simply curbs agitation and environmental stressors.
  • Supplementing with L-Theanine has been reported to improve relaxation, anxiety, and attention.
  • In an investigation examining caffeine, theanine, and the combination of the two, both supplements improved attention and alertness, yet the combination of caffeine and L-Theanine yielded the best results (Owen et al., 2008).

 

 

Pyridoxine HCl (15mg)

Pyridoxine is vitamin B6. B6 is an essential, water-soluble vitamin involved in many metabolic reactions as a coenzyme. Enzymes are made from protein, and a coenzyme is any non-protein molecule required for an enzyme to function properly. In this sense, coenzymes can “turn on” enzymes. In addition to its metabolic effects, B6 is also a coenzyme for hormone synthesis, and it helps boost mood and cognition.

  • Vitamin B6 is involved in the production of neurotransmitters, serotonin, dopamine, and GABA.
  • Low B6 levels are associated with depression and confusion, as well as anemia, weakened immune function, and mood disturbances.
  • Having low vitamin B6 levels doubles risk for the development of depression (Merete, Falcon, and Tucker, 2008).

FOCUS BLEND - 165mg

CDP Choline (100mg)

Choline is one of the most important nutrients for attention, focus, and cognition. Providing choline helps increase levels of acetylcholine, a primary neurotransmitter for cell-to-cell communication. These communications are literally what helps us think. It is considered the best form for improving brain function due in part to cytidine’s conversion to uridine – another ingredient in Phoenix Energy.

  • Only 2% of those who do not eat eggs get enough choline. Of those who do, only 57% do reach an adequate intake level for choline (Wallace and Fulgoni, 2017)
  • CDP Choline is involved in memory, neuroprotection, attention, and learning.
  • Healthy participants supplementing with CDP Choline experience improvements in processing speed, working memory, verbal memory, and executive function (decision making) (Bruce et al., 2014)

Uridine Monophosphate (50mg)

Uridine is a nucleotide found in RNA. Like other nucleotides, uridine interacts with receptors known as purine receptors (P2X and P2Y). Activation of certain purine receptors stimulates neuroprotection via release of nerve growth factor and other compounds. Uridine also promotes generation of phosphatidylcholine, which may improve the function of neurons.

  • Uridine contributes to phosphatidylcholine synthesis, which is both a source of choline and a significant component of neuron cell membranes.
  • Addition of uridine to the diet may enhance dopamine, boosting mood.
  • Perhaps the most important effects of uridine are on long term brain cell health, promoting dendrite outgrowth and synapse transmissions (Wang et al., 2005).

 

Niacin (16mg)

Niacin is vitamin B3, and essential vitamin. The primary function of niacin is to form nicotinamide adenine dinucleotide (NAD), a cofactor in metabolism that helps generate cellular energy as ATP. In part, the supply of ATP generated by NAD helps protect and fuel brain cells to promote memory and learning.

  • As a metabolic cofactor, niacin helps supply brain cells and other neurons with needed energy for proper functioning.
  • Supplying niacin may help with vascular and axonal remodeling.
  • Correcting niacin deficiencies may correct associated mental disturbances (Wang and Liang, 2012)

Huperzine A (150mcg)

Huperzine A is an acetylcholinesterase inhibitor commonly sourced from the Huperzia serrata plant. Acetylcholinesterase inhibitors reduce the rate at which acetylcholine is broken down, increasing available acetylcholine levels. Because acetylcholine is a major neurotransmitter, this is helpful for improving overall cognition and focus. Because Huperzine A reduce breakdown of acetylcholine and choline supplements, like CDP choline, Huperzine A and CDP Choline work synergistically, keeping your mind in a heightened state for an extended period of time.   

  • Huperzine A is special, as a specific acetylcholinesterase inhibitor, acting upon the G4 isoform of acetylcholinesterase – the form present in the mammalian brain.
  • Huperzine helps protect against over-excitation by glutamate, beta-amyloid, and peroxide.
  • Supplementing with Huperzine A has shown significant improvement in attention, cognition, and ability to multitask (Gul, Bakht, and Mehmood, 2018)

ENERGY BLEND

Citrulline Malate 2:1 (1,000mg)

1.       Pérez-Guisado, J., & Jakeman, P. M. (2010). Citrulline malate enhances athletic anaerobic performance and relieves muscle soreness. The Journal of Strength & Conditioning Research24(5), 1215-1222.
2.       Bendahan, D., Mattei, J. P., Ghattas, B., Confort-Gouny, S., Le Guern, M. E., & Cozzone, P. J. (2002). Citrulline/malate promotes aerobic energy production in human exercising muscle. British journal of sports medicine36(4), 282-289.
3.       Giannesini, B., Le Fur, Y., Cozzone, P. J., Verleye, M., Le Guern, M. E., & Bendahan, D. (2011). Citrulline malate supplementation increases muscle efficiency in rat skeletal muscle. European journal of pharmacology667(1-3), 100-104.
4.       Yabuki, Y., Shioda, N., Yamamoto, Y., Shigano, M., Kumagai, K., Morita, M., & Fukunaga, K. (2013). Oral L-citrulline administration improves memory deficits following transient brain ischemia through cerebrovascular protection. Brain research1520, 157-167.
5.       Lee, K. E., & Kang, Y. S. (2017). Characteristics of L-citrulline transport through blood-brain barrier in the brain capillary endothelial cell line (TR-BBB cells). Journal of biomedical science24(1), 28.
6.       Lee, K. E., & Kang, Y. S. (2018). l-Citrulline restores nitric oxide level and cellular uptake at the brain capillary endothelial cell line (TR-BBB cells) with glutamate cytotoxicity. Microvascular research120, 29-35.
7.       Kim, I. Y., Schutzler, S. E., Schrader, A., Spencer, H. J., Azhar, G., Deutz, N. E., & Wolfe, R. R. (2015). Acute ingestion of citrulline stimulates nitric oxide synthesis but does not increase blood flow in healthy young and older adults with heart failure. American Journal of Physiology-Endocrinology and Metabolism309(11), E915-E924.

Caffeine Anhydrous (280mg)

1.       Kennedy, D. O., & Scholey, A. B. (2004). A glucose-caffeine ‘energy drink’ameliorates subjective and performance deficits during prolonged cognitive demand. Appetite42(3), 331-333.
2.       Childs, E., & de Wit, H. (2008). Enhanced mood and psychomotor performance by a caffeine-containing energy capsule in fatigued individuals. Experimental and Clinical Psychopharmacology16(1), 13.
3.       Gupta, U. (1988). Personality, caffeine and human cognitive performance. Pharmacopsychoecologia.
4.       Childs, E., & de Wit, H. (2006). Subjective, behavioral, and physiological effects of acute caffeine in light, nondependent caffeine users. Psychopharmacology185(4), 514.
5.       Killgore, W. D., Rupp, T. L., Grugle, N. L., Reichardt, R. M., Lipizzi, E. L., & Balkin, T. J. (2008). Effects of dextroamphetamine, caffeine and modafinil on psychomotor vigilance test performance after 44 h of continuous wakefulness. Journal of sleep research17(3), 309-321.

Dynamine® (100mg)

1.       Kuhman, D., Joyner, K., & Bloomer, R. (2015). Cognitive performance and mood following ingestion of a theacrine-containing dietary supplement, caffeine, or placebo by young men and women. Nutrients7(11), 9618-9632.
2.       Wang, Y., Yang, X., Zheng, X., Li, J., Ye, C., & Song, X. (2010). Theacrine, a purine alkaloid with anti-inflammatory and analgesic activities. Fitoterapia81(6), 627-631.
3.       Compound Solutions, Inc. (2020) Pharmacokinetic Assessment of Methylliberine, Theacrine and Caffeine Interation*

Rauwolscine (1.5mg)

1.       Szabo, B., Hedler, L., & Starke, K. (1989). Peripheral presynaptic and central effects of clonidine, yohimbine and rauwolscine on the sympathetic nervous system in rabbits. Naunyn-Schmiedeberg's archives of pharmacology340(6), 648-657.
2.       Pettibone, D. J., Pfleuger, A. B., & Totaro, J. A. (1985). Comparison of the effects of recently developed α2-adrenergic antagonists with yohimbine and rauwolscine on monoamine synthesis in rat brain. Biochemical pharmacology34(7), 1093-1097.
3.       La Marca, S., & Dunn, R. W. (1994). The α-2 antagonists idazoxan and rauwolscine but not yohimbine or piperoxan are anxiolytic in the Vogel lick-shock conflict paradigm following intravenous administration. Life sciences54(10), PL179-PL184.
4.       Boonstra, E., de Kleijn, R., Colzato, L. S., Alkemade, A., Forstmann, B. U., & Nieuwenhuis, S. (2015). Neurotransmitters as food supplements: the effects of GABA on brain and behavior. Frontiers in psychology6, 1520.
5.       Smits, J. A., Rosenfield, D., Davis, M. L., Julian, K., Handelsman, P. R., Otto, M. W., ... & Powers, M. B. (2014). Yohimbine enhancement of exposure therapy for social anxiety disorder: a randomized controlled trial. Biological psychiatry75(11), 840-846.

MOOD BLEND

DL-Phenylalanine (400mg)

1.       Lehmann, W. D., Theobald, N., Fischer, R., & Heinrich, H. C. (1983). Stereospecificity of phenylalanine plasma kinetics and hydroxylation in man following oral application of a stable isotope-labelled pseudo-racemic mixture of L-and D-phenylalanine. Clinica Chimica Acta128(2-3), 181-198.

2.       Gold, M. S., Blum, K., Oscar–Berman, M., & Braverman, E. R. (2014). Low dopamine function in attention deficit/hyperactivity disorder: should genotyping signify early diagnosis in children?. Postgraduate medicine126(1), 153-177.

3.       Wood, D. R., Reimherr, F. W., & Wender, P. H. (1985). Treatment of attention deficit disorder with DL-phenylalanine. Psychiatry research16(1), 21-26.

4.       Beckmann, H., Strauss, M. A., & Ludolph, E. (1977). DL-Phenylalanine in depressed patients: an open study. Journal of neural transmission41(2), 123-134.

5.       Fischer, E., Heller, B., Nachon, M., & Spatz, H. (1975). Therapy of depression by phenylalanine. Preliminary note. Arzneimittel-forschung25(1), 132-132.


N-Acetyl L-Tyrosine (250mg)

1.       Neri, D. F., Wiegmann, D., Stanny, R. R., Shappell, S. A., McCardie, A., & McKay, D. L. (1995). The effects of tyrosine on cognitive performance during extended wakefulness. Aviation, space, and environmental medicine.

2.       Acworth, I. N., During, M. J., & Wurtman, R. J. (1988). Tyrosine: effects on catecholamine release. Brain research bulletin, 21(3), 473-477.

3.       Alonso, R., Gibson, C. J., Wurtman, R. J., Agharanya, J. C., & Prieto, L. (1982). Elevation of urinary catecholamines and their metabolites following tyrosine administration in humans. Biological psychiatry, 17(7), 781-790.

4.       Leyton, M., Young, S. N., Pihl, R. O., Etezadi, S., Lauze, C., Blier, P., ... & Benkelfat, C. (2000). Effects on mood of acute phenylalanine/tyrosine depletion in healthy women. Neuropsychopharmacology22(1), 52-63.

5.       Banderet, L. E., & Lieberman, H. R. (1989). Treatment with tyrosine, a neurotransmitter precursor, reduces environmental stress in humans. Brain research bulletin22(4), 759-762.


L-Theanine (90mg)

1.       Owen, G. N., Parnell, H., De Bruin, E. A., & Rycroft, J. A. (2008). The combined effects of L-theanine and caffeine on cognitive performance and mood. Nutritional neuroscience11(4), 193-198.

2.       Dodd, F. L., Kennedy, D. O., Riby, L. M., & Haskell-Ramsay, C. F. (2015). A double-blind, placebo-controlled study evaluating the effects of caffeine and L-theanine both alone and in combination on cerebral blood flow, cognition and mood. Psychopharmacology232(14), 2563-2576.

3.       Lu, K., Gray, M. A., Oliver, C., Liley, D. T., Harrison, B. J., Bartholomeusz, C. F., ... & Nathan, P. J. (2004). The acute effects of Ltheanine in comparison with alprazolam on anticipatory anxiety in humans. Human Psychopharmacology: Clinical and Experimental19(7), 457-465.

4.       Higashiyama, A., Htay, H. H., Ozeki, M., Juneja, L. R., & Kapoor, M. P. (2011). Effects of L-theanine on attention and reaction time response. Journal of Functional Foods3(3), 171-178.

5.       Ritsner, M. S., Miodownik, C., Ratner, Y., Shleifer, T., Mar, M., Pintov, L., & Lerner, V. (2011). L-theanine relieves positive, activation, and anxiety symptoms in patients with schizophrenia and schizoaffective disorder: an 8-week, randomized, double-blind, placebo-controlled, 2-center study. Journal of Clinical Psychiatry72(1), 34.

6.       Kimura, K., Ozeki, M., Juneja, L. R., & Ohira, H. (2007). L-Theanine reduces psychological and physiological stress responses. Biological psychology74(1), 39-45.


Pyridoxine HCl (15mg)

1.       Merete, C., Falcon, L. M., & Tucker, K. L. (2008). Vitamin B6 is associated with depressive symptomatology in Massachusetts elders. Journal of the American College of Nutrition27(3), 421-427.

2.       Skarupski, K. A., Tangney, C., Li, H., Ouyang, B., Evans, D. A., & Morris, M. C. (2010). Longitudinal association of vitamin B-6, folate, and vitamin B-12 with depressive symptoms among older adults over time. The American journal of clinical nutrition92(2), 330-335.

3.       Folstein, M., Liu, T., Peter, I., Buel, J., Arsenault, L., Scott, T., & Qiu, W. W. (2007). The homocysteine hypothesis of depression. American Journal of Psychiatry164(6), 861-867.

4.       Seshadri, S., Beiser, A., Selhub, J., Jacques, P. F., Rosenberg, I. H., D'Agostino, R. B., ... & Wolf, P. A. (2002). Plasma homocysteine as a risk factor for dementia and Alzheimer's disease. New England Journal of Medicine346(7), 476-483.

5.       Institute of Medicine (US) Standing Committee on the Scientific Evaluation of Dietary Reference Intakes. (1998). Dietary reference intakes for thiamin, riboflavin, niacin, vitamin B6, folate, vitamin B12, pantothenic acid, biotin, and choline. National Academies Press (US).

FOCUS BLEND

CDP Choline (100mg)

1.       Bruce, S. E., Werner, K. B., Preston, B. F., & Baker, L. M. (2014). Improvements in concentration, working memory and sustained attention following consumption of a natural citicoline–caffeine beverage. International journal of food sciences and nutrition65(8), 1003-1007.

2.       Wallace, T., & Fulgoni, V. (2017). Usual choline intakes are associated with egg and protein food consumption in the United States. Nutrients9(8), 839.

3.       McGlade, E., Agoston, A. M., DiMuzio, J., Kizaki, M., Nakazaki, E., Kamiya, T., & Yurgelun-Todd, D. (2019). The effect of citicoline supplementation on motor speed and attention in adolescent males. Journal of attention disorders23(2), 121-134.

4.       McGlade, E., Locatelli, A., Hardy, J., Kamiya, T., Morita, M., Morishita, K., ... & Yurgelun-Todd, D. (2012). Improved attentional performance following citicoline administration in healthy adult women. Food and Nutrition Sciences3(06), 769.

5.       Knott, V., de la Salle, S., Choueiry, J., Impey, D., Smith, D., Smith, M., ... & Labelle, A. (2015). Neurocognitive effects of acute choline supplementation in low, medium and high performer healthy volunteers. Pharmacology Biochemistry and Behavior131, 119-129.


Uridine Monophosphate (50mg)

1.       Wang, L., Pooler, A. M., Albrecht, M. A., & Wurtman, R. J. (2005). Dietary uridine-5′-monophosphate supplementation increases potassium-evoked dopamine release and promotes neurite outgrowth in aged rats. Journal of molecular neuroscience27(1), 137-145.

2.       Wurtman, R. J., Cansev, M., Sakamoto, T., & Ulus, I. H. (2009). Use of phosphatide precursors to promote synaptogenesis. Annual review of nutrition29, 59-87.

3.       Harms, K. J., & Dunaevsky, A. (2007). Dendritic spine plasticity: looking beyond development. Brain research1184, 65-71.

4.       Wurtman, R. J., Cansev, M., Sakamoto, T., & Ulus, I. (2010). Nutritional modifiers of aging brain function: use of uridine and other phosphatide precursors to increase formation of brain synapses. Nutrition reviews68(suppl_2), S88-S101.

5.       Richter, Y., Herzog, Y., Eyal, I., & Cohen, T. (2011). Cognitex supplementation in elderly adults with memory complaints: an uncontrolled open label trial. Journal of dietary suppleents8(2), 158-168.


Niacin (16mg)

1.       Wang, W., & Liang, B. (2012). Case report of mental disorder induced by niacin deficiency. Shanghai archives of psychiatry24(6), 352.

2.       Ye, X., Chopp, M., Cui, X., Zacharek, A., Cui, Y., Yan, T., ... & Chen, J. (2011). Niaspan enhances vascular remodeling after stroke in type 1 diabetic rats. Experimental neurology232(2), 299-308.

3.       Shehadah, A., Chen, J., Zacharek, A., Cui, Y., Ion, M., Roberts, C., ... & Chopp, M. (2010). Niaspan treatment induces neuroprotection after stroke. Neurobiology of disease40(1), 277-283.

4.       Yan, T., Chopp, M., Ye, X., Liu, Z., Zacharek, A., Cui, Y., ... & Chen, J. (2012). Niaspan increases axonal remodeling after stroke in type 1 diabetes rats. Neurobiology of disease46(1), 157-164.


Huperzine A (150mcg)

1.       Gul, A., Bakht, J., & Mehmood, F. (2018). Huperzine-A response to cognitive impairment and task switching deficits in patients with Alzheimer's disease. Journal of the Chinese Medical Association.

2.       Kozikowski, A. P., & Tueckmantel, W. (1999). Chemistry, pharmacology, and clinical efficacy of the Chinese nootropic agent huperzine A. Accounts of chemical research32(8), 641-650.

3.       Boudinot, E., Taysse, L., Daulon, S., Chatonnet, A., Champagnat, J., & Foutz, A. S. (2005). Effects of acetylcholinesterase and butyrylcholinesterase inhibition on breathing in mice adapted or not to reduced acetylcholinesterase. Pharmacology biochemistry and behavior80(1), 53-61.

4.       Ved, H. S., Koenig, M. L., Dave, J. R., & Doctor, B. P. (1997). Huperzine A, a potential therapeutic agent for dementia, reduces neuronal cell death caused by glutamate. Neuroreport8(4), 963-967.

5.       Wang, R., Yan, H., & TANG, X. C. (2006). Progress in studies of huperzine A, a natural cholinesterase inhibitor from Chinese herbal medicine 1. Acta Pharmacologica Sinica27(1), 1-26.

Q: What is the best way to use Phoenix Energy?

A: 15-30 minutes prior to work, study, or any other task demanding your complete attention.


Q: Is there anyone who should not use Phoenix Energy?

A: Those who are sensitive to caffeine, pregnant or nursing, taking any prescription medication, or are under the age of 18 should not use Phoenix Energy. Do not take Phoenix Energy prior to sleeping.